Tetanus. That year, a patient suspected of tetanus almost died in my hands.
I was particularly impressed that day on an emergency duty, although we were all atheists.
But it’s still a little pitiful to meet Ming, Chinese Yuan, etc., which means that it can be particularly busy.
It’s all good to be busy without a skin.
At 11:00 in the evening, the ambulance pulled back a patient.
It’s a young man, 32 years old, with a short chest of five hours, thinking it’s better to take a break at home, but it’s getting worse.
His wife called 120 and sent it to the hospital.
The doctor who left the car told me that he had made an EKG and that there was nothing unusual about it other than a CPR.
I saw the patient’s lips pale and a little obscuranty, and I quickly arranged for him to enter the rescue room.
Several nurses surrounded him, connected him to EK custody and opened a second vein.
Blood pressure is high and heart rate is high.
The point is that his blood oxygen saturation is only 94% (normally 98-100%) and it’s not normal in an oxygen-absorbed state.
You might ask, what does this data mean?
This means that the patient is either seriously inflammated by pneumonia or serious heart disease, such as serious myocarditis or heart infarction.
And these diseases, if they don’t pay attention, will kill him.
The patient is breathing, sweating, nervous, and speaking with a feeling of shaking, as if it was exhausting.
I’ll let him rest, let his wife speak for him, and I’ll have Dr. Phoe give him another EKG.
His wife told me that she had a fever after dinner this evening, and that she had to take her antics, but her chest was not well.
Then the chest became more and more uncomfortable, less and less breath, and a little bit of asthma and headache.
They are also more nervous, fearing that they will also be genetic, bearing in mind that the father of the patient died last year.
It’s not genetic, but since the patient’s father died because of it, it’s family history, so I can’t care.
In addition to making an EKG, get the nurse to give him blood, check on myocardial enzyme, mycium protein, sodium and conventional biochemicals.
I asked the patient if his chest was like a rock, and he couldn’t breathe and hurt.
He nods, whispers, yes, it hurts.
Dr. Pepe pulled out the EKT, and I grabbed it, and I looked forward to finding something, and I thought, “Don’t be so weird.”
If it’s still myocardial infarction, it might scare him to death.
Fortunately, the EKG did not find a typical change in ST-T, which means it may not be a heart attack.
But don’t be happy too soon, and I remind Dr. Phoebe that the EKG changes are time-sequenced, not necessarily a typical change in the first few hours of an infarction, a dynamic review, and another review in half an hour.
I gave him heart and lungs, and his heart beats very fast, except that there was nothing unusual about it, and there was a little moist noise in the lungs, possibly pneumonia.
The patient was very nervous, and I listened to the device near his chest, and I could scare him, and he was so tight.
I comforted him, not to worry. Even if you have a heart attack and come to the hospital, if you open the clogged veins in time, you’ll probably live well.
He nodded his head and whispered a thank you.
But I can still feel his muscles clattering and fighting the Cold War.
The nurse gave him sub-temperature, 38.9 degrees C, heat, no wonder he did.
Patients have visible chest asthma and fever, combined with the hysteria of their lungs, which is considered to be pneumonia and sepsis.
Next thing I know, I’m gonna have to get him to take a choreography or even pull him over and make a CT.
As soon as possible, because the patient’s blood oxygen is not very good, it’s difficult to fear that it will increase the lack of oxygen at any time.
The patient’s wife agreed to the chest CT examination, and I gave him a medical notification.
This kind of patient, with little attention, may be going to the ICT, and must communicate well in advance.
Then the nurse calls the emergency dispensary and issues the medicine, and antibiotics are the key.
The early use of antibiotics, the higher the survival rate, is a blood lesson.
Everything’s ready to go to the CT room, I’ll be with you.
But as we were about to launch the rescue room door, the patient suddenly shouted, “Don’t come over, go away.”
It’s not very loud. It’s not very clear, but it’s pretty gross.
The few of us had a moment and didn’t know what was going on.
The patient then said to the side, “Go away, you hear me?”
This time it was clear to everyone that a nurse looked pale and took a few steps back.
Honestly, I was scared too. But it soon came back. The patient must have been hallucinating.
There can’t be ghosts in the world, and it’s because of weather problems, not ghosts.
I told the next person that the patient was hallucinating and was about to prepare the midars and fluorine.
Midasron is a tranquilizer, and fluorine is an anti-depressant, anti-schizophrenic.
The patient’s face is weird. It’s probably not a joke. It’s just an illusion.
The CT room can’t go for a while.
The patient’s wife was in a state of panic, asking me what to do, and she was obviously scared.
I couldn’t comfort her because the patient’s condition had changed beyond my expectations.
I always thought he was a serious pneumonia, or had potential myocardial infarction, myocardial inflammation, etc., but these diseases could not be hallucinating.
Unless pneumonia is very severe, respiratory failure is severe, oxygen deficiency is severe, and oxygen deficiency does cause mental disorders and even hallucinations.
However, the lack of oxygen is not very serious, and the saturation of O2 has risen to 96 per cent after taking oxygen.
Cardiac and pulmonary disease does not explain his hallucinations.
I asked the family if there were any cases of schizophrenia in the patient’s home, such as grandparents, aunts and uncles.
The answer is no. Don’t tell me he doesn’t have a home, neither does his wife. Clean and white.
The family told me that the patient had been normal and had never been hallucinating, that sometimes there was more stress, because he was a programmer at a large factory, and he had a lot of work, but he was not hallucinating.
I can feel her going down.
I also noted the physical condition of the patient, whose physical condition has been so tight and tense since his admission.
I thought he was nervous at first, and now I have to suspect he’s out of his mind.
If the patient does not have a history of schizophrenia, it is important to consider whether there are brain problems, such as brain haemorrhage, brain tumors, etc.
In combination with fever, meningitis and meningitis are considered.
By the way, be wary of septic meningitis, Dr. Friede.
However, septic meningitis may have high-pressure manifestations within the skull, with severe vomiting, headaches, etc.
The patient’s wife told us he had a headache.
I stretched my hand to the neck, and he was very cooperative, and his neck was so stiff that I couldn’t get his neck back.
I let him relax again, but I tried, and his neck was still as hard as a wood, and he couldn’t bend.
“What’s this? I’m going to ask Dr. Frédé.
“The neck is strong. This means that the patient is stimulied and that there may indeed be meningitis or meningitis. I’m sorry.
Dr. Pepe looked up and seemed to have discovered a new continent and was very excited.
It’s not normal for me to look at the patient’s limbs, or to bandage them up and have a little muscle tension.
I looked specifically at whether the torso and limbs had rashes, but no, some sepsis patients would have typical rashes, and the diagnosis would have been clearer if they had found rashes.
But even in the absence of rashes, the above-mentioned objective evidence, particularly meningitis, may cause changes in body muscles, muscle tension, and the patients are hallucinating, and I can’t wait for the family to be the head CT.
If encephalitis or meningitis is misdiagnosed, it is different.
In particular, septic meningitis, which is good for children, is common among young people, and mortality and disability rates are high.
As soon as I think of this, my back is cold, and I’m going to have to do the head, the lungs, and the CT, and I’m going to ask the neurologist to come down.
Dr. Frepe is confused again. Is encephalitis, meningitis causing the patient to have a bad breath and a lack of oxygen?
Of course it’s possible, although not common, but it’s possible.
Respiration is brain-controlled. If there’s a brain problem, can you breathe alone?
I spoke to my family, saying that, in addition to pneumonia, meningitis and meningitis may be more critical.
Bacteria or viruses can enter the bloodstream from the respiratory tract and then enter the brain, causing cerebral inflammation, so that they can cause headaches, heat, a straight neck, increased muscle tension, even suffocation and oxygen deficiency may be associated with meningitis.
If not addressed in a timely manner, meningitis can be fatal.
From that point of view, I had the right notification.
And I’m right to have the antibiotics back in time, whether for pneumonia or meningitis, because most of the bacteria are bacteria.
Apart from CT, it’s more important to have vertebrae piercings, and to get brain vertebrae out of the test, if there’s actually bacteria or virus infection.
But it’s not appropriate for a patient to have a vertebrae puncture in an emergency, it’s too busy, and a vertebrae can’t do it in three or two, and I’m tired of having to wait until the neurological.
The family heard it was a head attack, and it just started crying.
I comforted her, and now it’s not time to cry, and I’ll do the CT first, then I’ll get a neurologist, early diagnosis and early treatment.
In addition to being nervous, without saying anything confusing, it seemed that the illusion was excessive and that the nurse would say thank you for giving him a shot, but still say the headache.
I’ll get the nurse to get the midarium, fluorine, and then go to CT.
In case he’s hallucinating or conceited, we may not be able to hold him off without the drugs.
Do CT when he’s scared and anxious.
I put his wife in a lead suit with him and comforted him.
I was focused on the CT screen.
The brain quickly echoes a series of manifestations of the patient, hemorrhaging, headaches, hallucinogenics, rapid breathing, oxygen deficiency, muscle stress, increased muscle tension…
Plus the death of the patient’s father from myocardial infarction, this is family history of cardiovascular disease, which is a brain haemorrhage, and that’s a big deal.
If you have a brain hemorrhage, you can’t just call a neurosurgery.
Just as I was thinking, the patient CT was finished.
At first sight, no brain bleeding, no brain infarction.
Not much unusual.
There are no signs of meningitis or meningitis. The lungs do have inflammation, but they’re not serious.
That makes me a little weird.
Maybe it’s early, even with meningitis or meningitis, the C.T. doesn’t necessarily see it.
I told Dr. Frepet that it’s definitely not serious pneumonia, that you look at the patient’s lungs, and they’re clean, so that they don’t cause so much breathing and suffocation.
Must be a brain problem.
Back to the rescue room, the patient’s blood-screeching results had come out, and the proxies were ticking.
The blood routine indicators are largely normal, and there are no anomalies.
I had Dr. Pippa pull his EKG again, and it’s still normal.
I can definitely rule out my heart infarction, and I’ll tell my family and the patient not to be too nervous.
We’re still looking at brain diseases, and we’re talking about neuron, and we’re getting paid for it.
The patient agreed to be hospitalized, whispered thirsty and wanted some water.
I saw his lips dry and sweated so much that the nurse filled him with two more bottles of liquid.
He was initially afraid of myocardial infarction or serious myocardial inflammation, afraid of rehydration, and afraid of increasing the heart burden.
But now that the calcium protein and myocardial enzymes are normal, there’s no heart failure, so there’s no chance of heart failure, so we’re gonna have to make up for it.
It’s probably too fast to drink. It’s probably shaking.
The patient’s scared, let his wife get her water.
“He’s afraid of water?” Dr. Freud looked at me.
I can’t help it.
He kept whispering to me if the patient was rabies.
rabies can be hallucinating, hot, suffocated, and watery.
It’s not that bad, is it?
If the patient’s behaviour is rabies, he/she is in a state of excitement, high excitement, fear, fear of water, fear of wind, rising body temperature, as well as larynx spasms, which makes it easy to drink water, although thirsty but afraid to drink it.
In addition, rabies can have vocal spasms, which can lead to a lack of sound and clarity.
In serious cases, there are hallucinations.
I’ve been wondering why he’s so obviously nervous, sometimes feeling muscle spasms.
If it’s rabies, it’s really bad.
I didn’t want to admit it, but I had to agree with Dr. Fupé.
Patients do have the possibility of rabies, because many of the symptoms are consistent, especially with the skull CT.
How I yearn for him to have a brain hemorrhage, or meningitis, which, though serious, is not necessarily fatal, and can live well with it.
But if it’s rabies, it’s gone.
I went to the patient’s wife and asked her quietly whether the patient had been bitten by a dog recently, for the last month, for the last, for the last, for the third, think about it.
She’s got no face, I don’t know what I’m asking.
I’m telling you, I suspect he has rabies and I want to know if he’s had contact with dogs or cats.
She had just cried, dryed her eyes and said to me, “No, it’s impossible, they never had a pet because they were pregnant and afraid of bad influence.
And the patients themselves are afraid of dogs, and they lived together for five years, and they’ve never provoked a dog or been bitten by a dog.
I have repeatedly verified that there is no dog bite history and no cat bite scratches.
They haven’t lived together in the last five years, five years ago, that’s hard to say.
I’ve grown so relieved.
Good.
Looks like it’s not rabies.
The rabies’ insulation period is usually just a few months, each for more than three years, and very few for more than five years. Although there are reports of more than 10 years, that credibility is lower.
So if a dog bites five years without a disease, it’s almost safe.
The patient hasn’t touched a cat or a dog in five years. That can’t be rabies.
Or think about the brain.
People don’t have to be afraid of water. Maybe they really don’t want it.
If he suffers from encephalitis, meningitis or an infarction, the damage to the brain tissue in a given area may also affect the throat muscles and lead to coughing.
This is common among some of the victims of brain infarction, who drink water and cough.
Patient’s blood pressure is still high, heart rate is still high, and I stepped up the rehydration.
The neurologist is here.
After listening to my medical history, I looked at the patient, read the CT film, and thought it was impossible to rule out meningitis and meningitis.
Not all meningococcal patients will be exposed to severe vomiting, depending on the internal pressure, and it is recommended that post-hospital vertebrae be improved.
And the skull CT has to be reviewed and the skull MRI has to be done. But it’s not tonight. It’s tomorrow.
The patient ‘ s family agreed to be hospitalized.
The patient is sober, and at this point there is no illusion, i.e. he is nervous and he does not talk, but he agrees to be hospitalized.
As soon as the neurologist left, the patient seemed to be getting upset. Say you’re going home.
“I thought we had a deal. I told him.
He doesn’t listen, his wife doesn’t talk. He keeps getting up and going home.
I want to take out the pin.
In fact, one of the pins had been pulled out by him, the nurse had been unable to stop it, the liquid from the infusion had quickly wetted the sheets and the needle on his back had started to bleed.
I ran over and held him down. I protected another vein.
“The patient may be pretentious or hallucinating. I told the nurse.
Mithalon is a tranquilizer that, in my experience, can calm the patient for seconds.
There are various risks to sedatives, but in order to be able to control him quickly, there is no way to do so.
Dr. Pepe and several other nurses came in to help control him.
When the nurse was preparing for the medication, the patient suddenly surged.
Like a patient with an epilepsy, he has lost consciousness, his eyes flipped, he shudders in his bed, and his muscles are tight.
It’s a hell of a thing to say. As soon as the neurologist left, the patient had the most serious cases of neuron and had an epilepsy.
“Come on, give him the mida palon. I’m pushing the nurse.
Midalium can stop this epilepsy in addition to calming down.
The patient’s wife was helpless, crying and crying, and the nurse had to get her out of the rescue room to spare us.
The medicine went down and the patient fell.
It’s only 10 seconds to sleep from twitch to quiet.
I was almost exhausted.
The worst thing to fear now is that the sedatives will keep the patient from breathing, because the patient is not breathing well, and the blood oxygen is barely strong, and if the breathing is inhibited and the oxygen is increased, it may have to go to the breathing machine.
I’ve got a lot of trouble, but I’m trying to keep my head down, stay calm, have Dr. Phoe call ICU and ask them if they have an empty bed.
If the patient’s in bad condition, maybe he won’t be able to get there.
It came back to me that the needle that the patient pulled out of the needle was bleeding and the sheets were red.
I also have some blood on me.
Why did the patient have an epilepsy?
It’s a further reminder that the stove is in the brain.
It’s either encephalitis, meningitis, or an infarction. After all, there’s been a change in the patient’s muscle and muscle tension.
The patient was quiet, but blood pressure followed, which was a side effect of sedatives.
It may also be that the patient was already under blood pressure, except that he was too nervous, causing false hypertension, so that the sedative calmed him down and the blood pressure showed itself.
I accelerated the rehydration, tried to increase the blood pressure, and asked the nurse to give him an additional pin.
One of these dangerous patients is not enough to have a retention needle and has to have two channels in order to be safe.
It’s like just now, if the patient has only one tunnel to refill it and it happens to be pulled out by the patient, it’s passive.
The patient’s wife asks me if it’s dangerous. She witnessed the rescue and was in shock.
I say yes, it should still be a brain problem, and if blood pressure and breathing don’t improve, maybe we should go to ICU.
As soon as she heard she was going to ICU, she broke up and fell on the floor crying.
I don’t know how to comfort her.
She said she wanted to see the patient.
I brought her into the rescue room.
She held the patient ‘ s left hand tight and shouted at him and cheered him.
I accidentally caught a little finger on the patient’s left hand and wrapped it in a sticker.
The years of clinical crawling and beating have given me a strange hunch, or fear of death.
I asked my family how this wound came from.
He said he accidentally got hurt while he was cleaning a grave in the country a week ago.
My throat was a little tight, and I asked exactly how it was hurt, whether it was stabbed with a knife or a woodcrumb.
I’m talking about tearing open the patient’s patch, not a big wound, a small mouth about a centimeter, but it’s a little dark and a little red and swollen.
Seeing this wound, I mentioned my voice.
And it’s also a great pain, so obvious, that I haven’t seen it for so long. I checked him back and forth several times. I didn’t notice the wound.
The patient’s wife told me that it was as if he had been cut by some tiles or stones and had shed some blood while he had weeded the cemetery and dug something in the dirt with his hands.
“Did you clean up after?” I asked her.
At the same time, a full body check was made to confirm that there were no other wounds.
“Simply water washes the wound and then puts a sticker on it. She told me.
“Is there any disinfection these days?”
“No, just a sticker. I’m sorry.
Oh, my God!
“Did he have a tetanus shot after that? I looked at her and said, “This is a crucial question. I’m sorry.
“None. “The patient’s wife is still naked.
Dr. Pepe also reacted. He was very nervous. “Is he tetanus? I’m sorry.
I slowly noded and endorsed Dr. Frépé’s judgment. And this judgment gave the patient’s family another blow.
She said she’d heard of tetanus before and that tetanus was as terrible as rabies.
Almost kneeling down and begging us to save her husband.
Tetanus. I didn’t think it would be tetanus. I’m not impressed because I haven’t seen a few tetanuss in clinical terms.
I always thought it was brain disease and even suspected rabies, but I didn’t think of tetanus alone.
Tetanus is an anaerobic (tetanus fungi) that can be neurologically pathological after reproduction. Where is the tetanus fungi? Some rusty props are found most often in the soil.
The victim ‘ s fingers are injured in the soil, which is quite likely to contain tetanus fungi, which quickly breeds when it enters the wound, especially in the case of small and long wounds, which can easily create an aerobic environment.
Tetanus fungus is anaerobic, and is the most aerobic, so the rate of reproduction is amazing. It also produces a large number of toxins, especially spasms.
Tetanus insulation periods average 7-8 days, or possibly within 24 hours, and the faster, the worse.
Patients suffer from symptoms of general inactivity, dizziness, headaches, chewing weakness, muscle tightness, and reflexes, typical of which are severe spasms based on muscle stress.
Usually the first to appear is a weak chewing muscle, difficulty in opening mouths, a constriction of mouths, a “smuggle” of mouths, a strong neck, and, if the back muscles are spasm, the torso of the patient is twisted into a bow, called the “horn bow back.”
With the exception of the horn bow, the patient is all in.
The patient was always nervous, I thought it was anxiety and fear, and then I thought it was brain disease, and I didn’t think it was tetanus.
And the patient whispers, doesn’t talk, drinks cough, which could be tetanus symptoms.
Thinking of here, my back was cold.
“Sensei, can a wound so small cause tetanus? Dr. Frepe looked at the patient’s wounds and asked me.
Of course it is possible, whether tetanus or not, that we have to inject him with tetanus toxoids and immunoglobins, although it may be a little late, but better than nothing.
At the same time, he must be completely sterilized with this finger.
I complain to my family that this wound should not have been so tight, nor should it have been so tight.
The patient’s family cried and asked me if it was tetanus.
I am not sure, but at present tetanus is most likely. His condition cannot be fully explained by meningitis or meningitis. Pneumonia is worse.
“Is there any way to get him diagnosed with tetanus, or do you want to take blood for antibodies or something? Dr. Frepe also asked me.
And that’s the biggest problem, and I told them that the most trouble with tetanus is that there’s no specifics, it’s hard to identify.
However, if the patient has an external injury, combined with muscle stress, mouth difficulties and a straight neck, it is almost.
If the patient has a horn bow, it’s certain. But not all patients are so typical.
“Do you notice that when the patient drinks water, his mouth is small, his neck is so hard, his limbs are hard, and he’s hallucinating, and he’s having a convulsion. The more I say it, the more excited I am, “a lot of small details that at first seemed insignificant and gradually replayed in my mind.” I’m sorry.
Get Dr. ICU to come and see if the patient is convulsed and can have respiratory muscles, muscular convulsions, severe oxygen deficiency or even death, bronchial cut-off or air-breathing.
All that can be done now is to support treatment of the disease, and antitoxin and immunoprotein are estimated to have been ineffective for too long.
The patient’s wife was hesitant to go to ICU, and I was in a hurry and I couldn’t deal with tetanus here, and when the bow was pumped back and forth, she had to use sedatives, which could keep breathing down.
Dr. ICTU’s coming, and we’re both thinking about it, and it feels like tetanus.
Convince the family and get ready for ICU.
Before I was on ICU, I completely disinfected his left finger, and the nurse gave him tetanus toxoids and immunoglobins.
When I handed the patient over to ICU, I finally got relieved and called the neurologist and said the patient was thinking about tetanus and went to ICU.
Then I learned that shortly after the patient went to ICU, there was a super-typical “bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow-bow.”
Tetanus patients are convulsed all over their muscles, their abdominal muscles may be convulsed, and when the back muscles are more powerful, when both muscles shrink, the body of the patient is twisted into a bow, as if it were a full bow, which looks terrible.
Since then, there has been no doubt about the diagnosis of tetanus.
Patients are not encephalitis, are not meningitis, are not brain haemorrhage or brain infarction, but tetanus.
It’s a shame that everyone’s busy trying to save their lives, and nobody’s thinking about taking pictures, otherwise it’s a very good picture.
Dr. ICTU used a lot of tranquillizers and accelerants to control the convulsions. At the same time, it was transferred to a single isolation ward to reduce light and sound interference.
The following day, the Director of the Section for Infections was called to visit for a comprehensive assessment and diagnosis of tetanus.
However, in view of the seriousness of the patient ‘ s condition and the risk of further severe convulsions at any time, particularly in the respiratory muscles, and the fear of the patient ‘ s aerobic suffocation, Dr. ICU has provided the patient with a bronchial cut to keep her breathing clear, which is the key to successful treatment.
The patient lived in the ICU for almost two weeks and was said to have experienced numerous convulsions later, but fortunately was able to be stopped in time and transferred to an infectious ward, where he stayed for more than a month and recovered.
Tetanus is also a very serious disease, and mortality rates are high, but not inevitable, and this requires positive and correct treatment.
Cope classroom: What are the daily injuries and tetanus alerts?
What is tetanus?
To put it simply, there is a bacteria (known as the tetanus fungi) infected with our wounds, which is anaerobic, and which does not like oxygen and, if the wound is thin and deep, it is very easy to create a deep oxygen-depletion environment, which is conducive to the survival and reproduction of the tetanus fungi, and, of course, the non-deep wound may be the survival and reproduction of the tetanus fungi.
A little tectonic concussion accelerates the production of many toxins, the most common of which is muscle stress and convulsions, the first of which starts with a sharp mouth chewing muscle, causing the patient to open his mouth, the “smuggle” with a strong neck, the back of his head, etc., and the muscles that are then exhausted are facial muscles, neck muscles, back muscles, abdominal muscles, limb muscles, and lastly muscular muscles, leading to breathing difficulties and suffocation.
Patients also suffer from fever, headaches and inactivity.
How long after the infection?
The average time is 7-8 days, which can be as short as 24 hours for several months and years, but generally around 7-8 days, the shorter the incubation period, the worse the condition, the worse the advance. Ninety per cent of patients have an illness within two weeks of injury.
What should we do with the wounds?
Because of the presence of tetanus fungi in the environment, especially in the soil, it is important to treat it when there is a wound, the principle being that it must be disinfected and, if the wound is deep, it must be sterilised and not covered directly by a sticker, which closes the wound, creates a relatively oxygen-depleted environment, and if it is infected, it can easily facilitate its reproduction.
Are you sure you’re gonna get tetanus?
Of course not, if it is common injury, without contact with soil, rust or something more dirty, there will normally be no tetanus fungi, and even if the soil is exposed, it will not necessarily have tetanus fungi, as long as the wounds are cleaned in time, and there will be no stress.
However, if the soil, rusty arrows or nails, wood crumbs, etc. are exposed, especially if the wound is more nuanced, it must be decontaminated and then administered to the hospital with tetanus antitoxin. The early application of antitoxins is very effective, but it is not good if it waits for the onset of the disease.
Is tetanus preventable?
Don’t get hurt, it’s best to prevent it.
It was also important that injuries should be fully and promptly recovered.
Anyone else ask if there’s a tetanus vaccine? Yes, our country has now promoted tetanus vaccination, which is planned and free of charge. But the antibodies produced by the tetanus vaccine, which last about 10 years, are not immunised for life, but are critical for neonatal tetanus prevention. Should adults be injured or dealt with in a timely manner, the injection should not be ambiguous. Case number: YX11PM7m4w
I don’t know.
Keep your eyes on the road.
