What kind of disease is hepatitis C? How can it cause hepatitis C infection?

What kind of disease is hepatitis C? How can it cause hepatitis C infection?

If we have a slightly older audience on the show today, we should remember that our Shanghai Hepatitis outbreak, which shocked the country in 1988, was due to the severe contamination of the Hepatitis Hepatitis virus, while the people of Shanghai ate it with open water, or even raw food, as was the case in the river basin. This resulted in a major outbreak of hepatitis A, with tens of thousands of patients per day, factory stoppages, school stoppages, and a series of severe injuries in the pre-spring provinces of Su, Zhejiang, Lu and Xuan as a result of the large number of exports to the provinces of Su, Zhejiang, Lu and Xuan in the pre-spring of 1988 and their sales on the spring market.

With the exception of hepatitis A and E, three other types of hepatitis B, C and D are similar, mainly mother-to-child transmission, sexual transmission and blood transmission, and sound familiar, like AIDS. Among them, hepatitis D is a defective virus that can rely solely on the symbioticity of Hepatitis B, so that hepatitis D is only likely to occur in patients already infected with Hepatitis B. Hepatitis B and C, in turn, are the worst players in hepatitis, characterized by high levels of infection, high susceptibility to transformation into chronic hepatitis and, ultimately, hepatitis cirrhosis and liver cancer, the two most common and lethal hepatitiss in the world today.

Of all these hepatitis types, hepatitis B is the most heard. China has the largest number of HB carriers and chronic HB patients in the world, with 130 million HB carriers, or 1/10 of the total population, that is to say, one in every 10 persons. Fortunately, most HB carriers do not have a lifetime disease, and even if they are converted to chronic HB, further transformation into cirrhosis and liver cancer can be prevented through good control. Speaking of the history of human struggle with hepatitis B, that’s another long story.

Now to the point, let’s move on to the main character of today, hepatitis C.

In fact, globally, the number of deaths from hepatitis B, which is the number one killer infectious disease in the United States and some countries in Europe, is far lower than that of hepatitis C, which is also the leading cause of liver cancer. Why is that? There are a few reasons, first of all because of the relative importance you’ve given to hepatitis B. Higher, hepatitis B has been screened at the time of medical examination, and hepatitis B vaccine has been developed successfully and began to be widely available in the 1990s.

And the Hepatitis C virus was not successfully isolated until the late 1980s. Moreover, with chronic hepatitis B without treatment, it usually takes more than 20 years to develop into liver cancer, even if some carriers do not have a lifetime disease. For hepatitis C, however, this period is much shorter and many patients have developed liver cancer 10 years after infection. Up to 20 years, the majority of hepatitis C patients develop from chronic hepatitis C to cirrhosis and liver cancer.

It is a terrible fact that, today, several decades after Hepatitis A vaccine has been developed, hepatitis C vaccine has been slow to develop and may never succeed. And that’s what this virus is about to say.

In fact, our country did not have any hepatitis C tests before 1994, so, more than a decade ago, in 2000 and a few years ago, many healthy people were suddenly diagnosed with hepatitis C during an accidental medical examination, and, one question being asked, all had blood transfusions in the late 1980s and early 1990s. The question is, what was the source of the large-scale Hepatitis C virus infection that year? Not really.

The reason for the large-scale transmission is not a single source, but rather the fact that persons who did not have any means of testing for blood transfusions were carrying hepatitis C. To date, however, more than 20 years have passed since hepatitis C was detected.

Imagine that if a person with hepatitis C who has not been discovered now gives blood, his blood could cause additional people to become infected with the virus, and these people will spread more to them, so the exponential rate of spread is inconceivable. By contrast, humans discovered hepatitis B virus in the early 1970s, almost 20 years before hepatitis C. This is also a major stumbling block to the elimination of hepatitis C in today ‘ s human race, where there may be tens of millions of uninformed HCV carriers in countries with a large population base, such as Pakistan and some countries in Africa, and where the number of people to be screened is extremely large.

This hepatitis virus, which was officially named by humans almost 30 years ago, is no worse than its brother, hepatitis A and B. Same

Hepatitis C virus has seven large groups of genes alone, and the difference between nucleic acids can be 30%! The magnitude and variability of the virus will allow the host’s immune defence system to surrender quickly. And even if hepatitis C virus is controlled by the use of anti-viral drugs, it can be re-emerged at any time as a result of the virus ‘ s supervariant properties, leading it to break through the immune system again. Therefore, the development of the hepatitis C vaccine is extremely difficult.

In the early stages of the discovery of the Hepatitis C virus, researchers had been helpless, as hepatic cells outside the body were unable to cultivate the virus and could only be used to study the replicability of the virus by infecting chimpanzees. Fortunately, the researchers soon found a breakout for hepatitis C.

First, the Hepatitis C virus is a hepatitis virus, and drugs can easily be circulated to the liver through oral administration. Second, the Hepatitis C virus is a RNA virus, and the entire process of self-replicating it will be carried out in the cell mass of the host, the human cell, without reaching the human cell core. So, theoretically, hepatitis C can be completely eliminated as long as appropriate drugs are found to suppress the replicability of the virus.

In contrast, hepatitis B is much more difficult because it’s a DNA virus that can enter the cell core of the host cell, entangled with the host’s DNA. And at this stage of human technology, there’s no way to get the drug to the cytological nuclei and eliminate the entanglement of HB virus DNA. So, for hepatitis B virus, the drug inhibits the replica of the virus, but DNA information remains in the cell core. Once there is a chance, it will come back.

In order to help you better understand the mechanisms in which these viruses work, we first need to learn some basic biological knowledge.

Each base can match another base, A always pairs with T, G total pairs with C. The full name of RNA is nucleic acid, which is a “deoxy” word less than DNA, which by definition is an oxygen atom less than DNA, consisting of a nitrogen-containing alkalin + nucleotide + phosphoric acid, and the base that makes up RNA is different from DNA, which is: gland A, bird luminum G, urine U, cytex C, a t, and, again, DNA is AGTC, and RNA is AGUC. So, we can find that DNA and RNA are really, like, a little bit less of a molecular structure, but this little difference in structure ultimately has a very different effect.

DNA exists in the cell core, and RNA exists only in the cell mass, which is the intermediate that DNA produces in the expression of protein. It’s like, for example, DNA, which is like a real-season dress in a LV shop, and RNA, which is like a house price made by the owner of a treasure shop, which at first glance is the same, but the details are always different, and the metaphor is, of course, Vivi came up with it, not me, and I’ve never been in a LV shop.

You can only copy the instructions word by word in paper and pen, which is called a transliteration, and you copy the manuscript, with individual letters that differ from the original manuscript, but the overall message is the same, which is called the messenger RNA, or mRNA, or m-message in English. You take your copy of the manuscript home, and you match it with a piece of it with a piece of it called a translation, and eventually you get a part of it that’s assembled, and that’s different amino acids, and these amino acids are organized in different forms of chemistry, forming a protein molecule with different structures and functions.

The proteomic molecules, in turn, form a single cell in the human body through various forms of integration, and eventually collide the human body with this complex and sophisticated instrument.

So what exactly is the virus? The virus is not a cell; it does not have a complete structure such as a membrane, a cell mass, a cell nucleus, which contains only a single nucleic acid non-cellular micro-organisms and must be parasitic within the living cell. In other words, whatever the virus is, it must live in the host’s cell. Hepatitis C virus is a virus that contains only a single chain of RNA (i.e. a chain structure) as a genetic material. Many of the large-scale infectious diseases that we know have been endemic to people, such as SARS more than a decade ago, Ebola in Africa, influenza A, H1N1, avian influenza, and so on, are RNA viruses.

The biosynthesis of the RNA virus is extremely unique, and when the human body is infected with the Hepatitis C virus, the virus transmits the liver to the blood as a fat particle, which is a Trojan horse, spreads like cells in all parts of the body, and enters the cell form through internal ingestion in association with a receptor in the cell membrane, where it is like being eaten by a membrane.

This copying process helps you understand, like a page in your hand, you want a page on the copier. The so-called structural protein is the paper required for the reproduction; not the structural protein is the photocopying machine. With polypolase, the virus RNA, like DNA double-chain structure, synthesizes a complementary RNA chain with a base pair, then untangles it into a generation RNA chain. Thus, the destruction of protein enzyme formation is a key link in inhibiting the self-replicating of hepatitis C virus. In short, it was the destruction of a photocopying machine, so that a new page could not be copied.

Here, it has to be said that there is another virus, similar to the supertransformative properties of Hepatitis C, that is, HIV – HIV – which everyone smells like. Extremely variable viruses are characterized by their ability to successfully bypass the human immune system, their vulnerability to drug resistance and their extreme difficulty in developing vaccines.

And as the two ultimate killers that plague human life, the HIV virus is much more powerful than the RNA virus, but it’s a retrovirus that can become a DNA integrated into the host’s chromosome, and the host cell is completely incapable of removing the integrated virus. And that’s why HIV can’t be cured, and 100 percent of people are infected with HIV for life, and no one can get rid of it.

There is no reason why some alternative medicine should be presented as a cure, even if it is not clear. How can it be addressed? For example, thousands of years of simple philosophical thinking and therapeutic practice have failed to find a cure for smallpox, and modern medicine has learned the root causes of smallpox and developed a vaccine that will make the smallpox virus disappear from the planet.

What kind of disease is hepatitis C? How can it cause hepatitis C infection? – The response recommended by the salt selection.

I don’t know.

Keep your eyes on the road.